I am a PhD candidate with a strong research profession. I have been working on my PhD at George Mason University in Bioinformatics, and Computational Biology, and earned my Master of Science (M.S.) focused in biotechnology from University of Maryland University College.

I have worked in the laboratory setting in the healthcare system for 4 years, in academia for 2 years and in industry for 3 years.

I have achieved my post-Master’s Fellowship at National Cancer Institute in Dr. Mark Lewandoski’s lab. During this fellowship, I performed many biochemical and molecular techniques including PCR, quantification of mRNA (qPCR) and protein expression, immunohistochemistry, in situ hybridization based on the mechanism of the hybridization chain reaction (HCR), Western blot, advanced molecular imaging and analysis, to elucidate the signaling requirements for kidney development, with a focus on FGF signaling.

I have been working on many projects as associate scientist at Meso Scale Diagnostic (MSD). During my time at MSD, I have been generating data while using a wide range of MSD electrochemiluminescence immunoassays and analyzing data by using appropriate software, including statistical analyses, and preparing tables/graphs with R-Studio to highlight findings. Two of these projects have been published with the following titles:

Viral Antigen and Inflammatory Biomarkers in Cerebrospinal Fluid in Patients With COVID-19 Infection and Neurologic Symptoms Compared with Control Participants Without Infection or Neurologic Symptoms

Patients with Severe COVID-19 Do Not Have Elevated Autoantibodies Against Common Diagnostic Autoantigens

Currently, I have been working on my dissertation project on inflammatory signaling pathways in ulcerative colitis. I have been using bioinformatics tools to identify the biomarkers that can be UC indicators, verify the Genetic causes of UC, identify the cell signaling that are involved in underlying mechanisms of UC, find the pathways connected to inflammatory responses of T cells, and find epigenetic causes of UC.